Myocardial protection with current methods of cardioplegia is still incomplete 1, 2, 3. Several studies have shown that hyperkalemic solution may lead to cell injury 1, 2. Adenosine-lidocaine-magnesium (ALM) is a new normokalemic, “polarizing” method of cardioplegia that arrests the heart at its resting membrane potential (1). Pre-clinical studies have shown ALM to provide improved protection compared with hyperkalemic solutions 1, 2. A recent clinical trial also observed improved outcomes with ALM-enriched hyperkalemic cardioplegia (3). We hypothesized that normokalemic, “full-polarizing” ALM microplegia from induction to reanimation provides improved myocardial protection compared with hyperkalemic arrest (3).